Dream picture’s Logo 2011

Posted août 30th, 2011 by coyote37

Here the sum of logos animmated by dream picture:

Dreampicture demoreel 2011.

Posted juin 20th, 2011 by coyote37

Hi guys! hope your well!

Here it is! the demoreel from Dreampicture for 2011.

Hope you’ll enjoy!

have a good day!

coyote.

CV from the man behind dreampicture.fr!

Posted mai 30th, 2011 by coyote37

Hi guys! hope your weeeeeeeeeeeelll long time noo news…. sooooooooooooo sorry.

BUT…. i was working on that:

Hope you will enjoy ths particlar CV presentation!

have a good day!

coyote.

BOT and DREAM

Posted février 11th, 2011 by coyote37

Hi everynone! long time no tutorials i know but currently working for the next one!

so here a preview for the next one:

i’ll show yo how i did this WP using Cinema 4D and after effects. People don’t work often on one frame and that’s will be the point: starts from frame and then animate!

and a quick render after my MILG8 training, enjoy and thanks robert redman ( see in links):

Have a nice evening and take care of your self!

coyote

The flower out!

Posted février 2nd, 2011 by coyote37

Finally i finished my flower with trapcode form!

here it iiiiiiiiiiiiiiiiiiiiiiiiiiiis:

have a nice day!

 

 

 
 
 
It is a very excellent animation which explains the hiv replication very clearly.
For free download of this video please visit my webpage
http://3dmed.wetpaint.com/
And other 3D animation videos visit
http://3dmed.wetpaint.com/page/3D+Medical+Animation+Library
Regards,
Dr.Rufus

The Lyrics of this video is here

Targeting HIV replication

The replication of HIV 1 is a multi-stage process.

Each step is crucial to successful replication and is therefore a potential target of antiretroviral drugs.

Step one is the infection of a suitable host-cell, such as a CD4-positive T-lymphocyte.

Entry of HIV into the cell requires the presence of certain receptors on the cell surface, CD4 -- receptors and co-receptors such as CCR5 or CXCR4.

These receptors interact with protein-complexes, which are embedded in the viral envelope.

These complexes are composed of two glycoproteins:

an extracellular gp 120 and
a transmembrane gp 41

When HIV approaches the target cell gp120 binds to the CD4-receptors. This process is termed attachment.

It promotes further binding to a co-receptor. Co-receptor binding results in a conformational change in gp120.

This allows gp41 to unfold and insert its hydrophobic terminus into the cell membrane.

Gp 41 then folds back on itself.

This draws the virus towards the cell and facilitates the fusion of their membranes.

The viral nucleocapsid enters the host cell and breaks open releasing two viral RNA-strands and 3 essential replication enzymes:

Integrase, Protease and Reverse Transcriptase.

Reverse Transcriptase begins the reverse transcription of viral RNA.

It has two catalytic domains:

The Ribonuclease-H active site

And the polymerase active site

Here single stranded viral RNA is transcribed into an RNA-DNA double helix. Ribonuclease- H breaks down the RNA.

The polymerase then completes the remaining DNA-strand to form a DNA -- double helix.

Now Integrase goes into action.

It cleaves a dinucleotide from each 3-prime end of the DNA creating two sticky ends.

Integrase then transfers the DNA into the cell nucleus and facilitates its integration into the host cell genome.

The host cell genome now contains the genetic information of HIV.

Activation of the cell induces transcription of proviral DNA into messenger RNA.

The viral messenger RNA migrates into the cytoplasm where building blocks for a new virus are synthesised.

Some of them have to be processed by the viral protease.

Protease cleaves longer proteins into smaller core proteins.

This step is crucial to create an infectious virus.

Two viral RNA-strands and the replication enzymes then come together and core proteins assemble around them forming the capsid.

This immature particle leaves the cell acquiring a new envelope of host and viral proteins.

The virus matures and becomes ready to infect other cells.

HIV replicates billions of times per day destroying the hosts` immune cells and eventually causing disease progression.

Drugs which interfere with the key steps of viral replication can stop this fatal process.

Entry into the host cell can be blocked by fusion inhibitors for example.

Inhibition of reverse transcriptase by nucleoside inhibitors or by non-nucleoside Reverse Transcriptase- inhibitors is part of standard antiretroviral regimens.

The action of Integrase can be blocked.

Protease inhibitors are also part of standard antiretroviral therapy.

Each blocked step in viral replication is a step towards better control of HIV disease.


Script, Storyboard, Art Direction by: Frank Schauder, MD
Animation: MACKEVISION
Publicity: Dr.Rufus Rajadurai.MD.,D.DiaDENS.,

 

 

 

 

 

 
 
 
 
 
 

+ Recent posts